Jump to content

Sponsors

VALOR

Members
  • Posts

    76
  • Joined

  • Last visited

Posts posted by VALOR

  1. Yeah I meant on a 10 panel drug test will it produce a false positive for amphetamines? The chemical structure is similar to amphetamines so in theory it could cause someone to have a positive urine analysis. I would rather not invest in a product and then find out I cannot use it. Thank you! Btw I edited the post I previously made. I think autocorrect may have misled some people.

  2. if anybody knows a good doc in nc/ raleigh area please pm me. my current doc is dragging his feet because my levels are 390 in the morning. they drop to 200 in the afternoon but he claims this is normal! He apparently doesnt understand the hpta. thanks in advance

  3. 1biggun11 you are on the right track. my natural levels are just above the minimum for hrt. this would only be a temporary thing. im really just wondering how long it will take for hpta to be shut down and if my ng/dl will be in the single digits.

  4. I have questions regarding aromasin's mechanism of action. I read that a daily dose of 25 mg aromasin effectively suppresses 70 percent of estrogen production in non steroid using males. Would more aromasin be needed relative to the quantity of exogenous test used or does the body have a set limit of aromatase in the body regardless of hormone levels?

    Are aromatase enzymes rendered ineffective indefinitely by aromasin? Does the body have to produce more aromatase now? How long would it take for aromatase to be replenished if this is the case? I'm curious about this ai because I was an over responder to arimidex.(headaches joint pain nausea) I realize that everyone will respond differently and practical experience is paramount along with bloodwork. I am mainly concerned about the proper dosing protocol. Does the drug bind steadily to aromatase over the active life of the drug or does aromasin immediately bind to aromatase and exert its effects via the lack of aromatase while the body replenishes its levels over a few days? If it is a steady decrease all I would need is a dose every few days but if it's immediate a smaller daily dose is required. Basically I'm trying to avoid severe fluctuations in estrogen levels.

    Does anybody have blood work from daily administration or bi-weekly administration? How do hrt doctors use this drug alongside hrt?

  5. I have been looking into the half lives associated with esters and I don't quite understand how increasing a dose changes the percentage of hormone released. For instance if I injected 25 milligrams of tren ace one time there would be 12.5 milligrams left of esterfied compound after 72 hours. Therefore, the body would be converting 4 milligrams a day to active trenbolone base. Yet, if I inject 100 mgs of tren ace there would be 50 mgs of esterfied compound left in 72 hours. So the body would be converting 15 mgs on average a day according to the accepted half life of the acetate ester. What is the explanation for the change in how much of the compound is utilized over 72 hours. I assume that an injection creates a depot regulating how much access the blood has to the hormone consequently regulating conversion of bound compound to active compound. What do you believe is responsible for the difference in change? Is the hormone absorbed into the blood completely following an injection or does it remain within the muscle? It would make more sense if there were designated percentages of how much compound is released relative to time. In my search to discover the answer I came across a chart on revalor administration to cattle. Cattle are injected in the subcutaneous area of the ear between blood vessels allowing for trenbolone acetate to be released for up to 100 days with a single dose ranging up to 200mg. http://www.pbsanimalhealth.com/implantchart

    This suggests a steady rate of release although they are trenbolone acetate pellets.

  6. So I found the answer to my own question...thank you for all the input on what you think of the compounds in respect to what you employ them for...proviron is rendered inert before it can have any effect on the ar by the hydroxysteroid dehydrogenase. Masteron on the other hand has a chemical alteration allowing it to bind to the ar. Therefore masteron is a more effective agent than proviron since it is not converted into an inactive hormone and does bind to the at.

  7. i read that dht, specifically in an article about proviron, is rendered inert in the body if too much is present in the body. is this true about masteron or is it chemically modified enough to exert beneficial effects in high dosages?

  8. How would you reccomend using insulin to maximize glycogen retention before an intense cardio session like a long run? Could insulin be used for a short duration safely to improve performance? How does short term insulin use for an event affect insulin sensitivity and natural secretion?

×
×
  • Create New...